Joint & Musculoskeletal Conditions

Why chronic pain is more than wear and tear

Inflammation does two jobs, and the second one is pain

Most people understand inflammation as the thing that damages tissue. It is, but that is only half of what it does.

Inflammatory messengers, particularly TNF-alpha and IL-6, are also direct pain amplifiers. They lower the threshold at which nerves fire, so an ordinary sensation becomes uncomfortable and an uncomfortable one becomes severe. A review of inflammatory biomarkers in pain found that levels of TNF-alpha and IL-6 correlate directly with how intense people’s pain is and how often it occurs.

This is the single most useful fact on this page, because those same two messengers respond to diet. A systematic review and meta-analysis of randomized controlled trials found low-carbohydrate diets significantly lowered TNF-alpha and IL-6. So there is a direct chain here: change what you eat, lower the messengers, and turn down both the tissue damage and the volume of the pain signal.

Part of how that happens is metabolic rather than dietary in the narrow sense. When your body shifts toward burning fat it produces beta-hydroxybutyrate, a ketone body that directly inhibits the NLRP3 inflammasome, one of the master switches your immune system uses to generate inflammatory signals in the first place.

Your pain system can turn its own volume up

Pain that continues for months changes the nervous system that carries it. Nerve pathways become more efficient at transmitting pain signals, and the brain regions processing them become more reactive. Clinicians call this central sensitization, and the practical consequence is that pain can become somewhat independent of the original injury.

This is the explanation for a lot of things that otherwise look inexplicable: pain that spreads beyond where it started, pain that outlasts the healing, pain in people whose scans look unremarkable. It is why being told your imaging is normal is not the same as being told nothing is wrong.

It matters for treatment because sensitization is fed by ongoing inflammatory signalling and by poor sleep. Both of those are addressable. Reducing the inflammatory input reduces the pressure keeping the system turned up.

Body weight loads your joints twice

Excess weight does the obvious thing, which is put mechanical load on the spine, hips, and knees. It also does something less obvious and arguably more damaging: fat tissue is metabolically active and secretes inflammatory signalling molecules into circulation. A review in Nature Reviews Rheumatology describes how these promote low-grade inflammation and the breakdown of connective tissue, and identifies disc degeneration as a common comorbidity of obesity and type 2 diabetes for that reason.

So weight is not just a load problem. It is an inflammation problem wearing a load problem’s clothing, which is why losing it tends to reduce pain more than the change in pounds alone would predict.

Sleep sits in the middle of the circuit

Pain wrecks sleep, and poor sleep lowers your pain threshold the next day. It also raises inflammatory markers directly. That is a self-reinforcing loop, and it is the reason so many people with chronic pain describe a slow deterioration rather than a steady state.

Sleep is one of the first things our patients report improving, often before pain does. It is worth knowing that, because it is also the thing that starts unwinding the loop.

What that means for treatment

Take out the foods provoking an immune response. Lower the inflammatory messengers that both damage tissue and amplify pain. Reduce the metabolic inflammation coming from excess weight. Improve sleep. Then reintroduce foods one at a time so you learn precisely what your own body reacts to.

None of that replaces what your rheumatologist or your spine specialist does. It addresses a layer that usually goes untouched.

Rheumatoid Arthritis

What rheumatoid arthritis is

Rheumatoid arthritis is an autoimmune condition in which the immune system attacks the synovium, the lining of the joints. It usually affects both sides of the body symmetrically and starts in the smaller joints of the hands and feet before moving outward.

What defines the daily experience is less the pain itself than the stiffness and the fatigue. Mornings are the worst part for most people, and the tiredness is not the kind that rest fixes.

How rheumatoid arthritis affects your day

• Morning stiffness lasting well over half an hour, sometimes hours
• Swollen, warm, painful joints, usually matching on both sides
• Losing grip strength: jars, keys, buttons, taps, bottle caps
• Fatigue heavy enough to shape what you agree to do
• Flares that arrive without an obvious reason and clear the same way
• Feeling unwell and feverish alongside the joint symptoms
• Dry eyes and mouth
• Gradual loss of what you can physically do, which is often the part people find hardest

What is driving the pain

• Inflammatory messengers amplifying the signal. TNF-alpha and IL-6 both erode the joint and make the nerves around it more sensitive. This is why the pain of rheumatoid arthritis is often out of proportion to what imaging shows, and why bringing those messengers down helps faster than joint repair could.
• The gut connection. Increased intestinal permeability is documented in rheumatoid arthritis, and immune activation appears to begin at mucosal surfaces long before joints are involved. It is a large part of why removing dietary triggers has an effect at all.
• Specific food triggers. Many people with rheumatoid arthritis have foods that reliably worsen their symptoms. Which foods varies enormously, which is why generic advice underperforms and identifying yours matters.
• Metabolic inflammation. Fat tissue secretes inflammatory signals, and higher body weight is associated with more disease activity and a poorer response to medication.
• Sleep disruption and central sensitization. Both common in rheumatoid arthritis, and both raise the pain you feel from the same amount of joint inflammation.
• Smoking. The strongest known environmental risk factor, and associated with more aggressive disease.

How Revero treats rheumatoid arthritis

This is the condition where both halves of our approach have been tested most directly.

In the MIKARA trial, a double-blind randomized controlled study of 61 rheumatoid arthritis patients on stable medication, the group put into ketosis saw a significant reduction in disease activity across 16 weeks while the control group saw none, with improvements specifically in pain, morning stiffness, and quality of life. A 2026 study of an elimination protocol in rheumatoid arthritis found improvements in disease activity, pain, fatigue, and sleep in most participants within eight weeks. And a review of low-carbohydrate approaches in inflammatory arthritis found they reduce inflammation and ease joint symptoms.

Your plan works on both fronts at once: removing the foods provoking immune activity and taking pressure off the gut barrier letting them through, while the shift in your metabolism suppresses the inflammatory switch producing the cytokines that are damaging your joints and sensitising your nerves.

Your provider follows hs-CRP and a full panel at baseline and every three months, so whether this is working gets an objective answer rather than depending on how your hands felt this morning. You log joint pain, swelling, how many joints are involved, morning stiffness, and fatigue daily, on the instruments a rheumatologist would use.

Your coach handles something that sounds small and is not: preparing food when your hands hurt. Simple preparation, batch cooking on the better days, and practical ways to hold the plan through a flare. As your disease activity and markers come down, your provider can begin reducing medication, gradually and in coordination with your rheumatologist.

Among Revero patients with rheumatoid arthritis, disease activity improved 40.5% in three months.¹ In an independent Harvard Medical School affiliated study, 96% of participants with musculoskeletal conditions reported their condition resolved or improved.³

Ankylosing Spondylitis

What ankylosing spondylitis is

Ankylosing spondylitis is inflammatory arthritis of the spine and the sacroiliac joints where the spine meets the pelvis. Over years, sustained inflammation can drive new bone formation and reduce how much your spine can move.

It has a pattern that distinguishes it from ordinary back pain, and it is worth knowing because it is frequently mistaken for a mechanical problem for years. The pain is worse after rest and better with movement. It wakes people in the second half of the night. And it usually starts before the age of 45.

How ankylosing spondylitis affects your day

• Back and buttock pain that eases once you get moving and returns when you stop
• Waking in the early hours and needing to move to settle
• Morning stiffness lasting more than half an hour
• Heel pain, or pain in the chest wall when breathing deeply
• Losing range of motion gradually: reaching, turning, bending
• Fatigue that tracks with how active the disease is
• Painful red eyes with light sensitivity, which needs prompt attention
• Digestive symptoms, which are more common in this condition than most people are told

What is driving the pain

• The gut-spine connection. This is the most important and least discussed feature of ankylosing spondylitis. A large proportion of patients have microscopic inflammation in the intestine despite having no digestive complaints at all, and a meaningful subset go on to develop inflammatory bowel disease. The relationship between gut inflammation and spinal inflammation here is well established, and it is the clearest reason a gut-directed approach makes sense.
• Inflammatory messengers. The same cytokines driving the enthesitis and spinal inflammation also sensitise the nerves reporting it, which is why disease activity and pain do not always track imaging.
• Excess weight. Associated with greater inflammation, more severe disease, and higher cardiovascular risk in ankylosing spondylitis specifically.
• Night pain and sleep disruption. A defining feature of this condition, and a driver of next-day pain sensitivity in its own right.
• Deconditioning. Pain reduces movement, and reduced movement worsens stiffness and pain. In a condition where movement genuinely helps, this loop is costly.
• Genetics. HLA-B27 is present in most people with the condition, though most carriers never develop it. Something has to trigger it.

How Revero treats ankylosing spondylitis

If your spine is inflamed and your gut is quietly inflamed too, treating only the spine addresses half of it.

Removing the foods that increase intestinal permeability and provoke immune activity reduces the inflammatory load coming out of the gut, and that holds whether or not you have any digestive symptoms. Lowering carbohydrate reduces the inflammatory contribution of excess weight, which matters more here than in most conditions, and raises ketone levels that suppress inflammatory signalling directly. Randomized trial evidence shows low-carbohydrate diets significantly lower TNF-alpha and IL-6, the messengers that both drive this disease and amplify the pain it produces.

Your provider follows hs-CRP and a full panel at baseline and every three months, which matters in a condition where inflammation does structural damage over years that you cannot feel happening. Daily, you track back pain, other joint pain and swelling, and how long morning stiffness lasts, which is usually the earliest sign the plan is working.

Because movement is genuinely part of treatment here, your coach works on keeping you consistent with it rather than treating it as separate from the nutrition side, and on protecting your sleep, since night pain and next-day pain feed each other. As your symptoms and markers improve, your provider can begin reducing medication, gradually and with your consent.

In our survey of more than 11,000 people following a low-carb high-protein diet, 91% of those with joint and musculoskeletal conditions reported improvement.² In an independent Harvard Medical School affiliated study, 96% of participants with musculoskeletal conditions reported their condition resolved or improved.³

Lupus

What lupus is

Systemic lupus erythematosus is an autoimmune condition in which the immune system produces antibodies against the body’s own tissue. It can affect many systems at once, which is why it varies so much between people and takes so long to diagnose.

For most people with lupus, the two symptoms that dominate daily life are joint pain and fatigue. The joint involvement is usually in the hands, wrists, and knees, and it tends not to cause the erosion that rheumatoid arthritis does, which sometimes leads to it being taken less seriously than it deserves. The fatigue is frequently the most disabling part of the entire condition.

How lupus affects your day

• Fatigue that is not proportionate to what you did and does not resolve with rest
• Joint pain and swelling, commonly in the hands, wrists, and knees
• Morning stiffness
• A rash across the cheeks and nose, and rashes elsewhere triggered by sun
• Fever without infection
• Mouth or nose ulcers, and hair thinning
• Chest pain on deep breathing
• Difficulty concentrating and holding attention
• Flares that make planning anything feel unreliable

What is driving the pain and fatigue

• Systemic inflammatory signalling. Lupus produces inflammation throughout the body, and those messengers both cause the joint symptoms and directly generate the fatigue and cognitive fog. That is why fatigue in lupus is a physical symptom rather than a mood or a motivation problem.
• Gut barrier function and the microbiome. A growing body of research links intestinal permeability and microbiome composition to lupus activity, following the pattern seen across autoimmune disease.
• Central sensitization. Widespread pain is common in lupus and is often out of proportion to measurable joint inflammation, a hallmark of an amplified pain system.
• Sleep disruption. Extremely common in lupus, and a direct contributor to both next-day pain and next-day fatigue.
• Ultraviolet light. A well-established trigger for flares. Sun protection is genuinely part of treatment.
• Hormones and genetics. Far more common in women, particularly during the reproductive years, with multiple immune-regulating genes implicated.

How Revero treats lupus

Most lupus treatment works by suppressing the immune response. Very little of it addresses what keeps provoking that response in the first place, or does anything about the fatigue.

Our approach works on both. Removing the foods driving immune activation and increasing intestinal permeability reduces the inflammatory input your body contends with every day. Lowering carbohydrate raises ketone levels that suppress inflammatory signalling directly and reduces the contribution of excess weight and insulin resistance. Randomized trial evidence shows low-carbohydrate diets significantly lower TNF-alpha and IL-6, two of the messengers central to lupus activity and to the fatigue it causes.

Monitoring is more thorough in lupus, deliberately. Your provider follows hs-CRP, kidney markers, and a complete blood count at baseline and every follow-up, because lupus can move quietly and the labs often speak before the symptoms do. You track rash, joint pain, joints involved, and fatigue daily.

Your coach’s focus here tends to be energy: building a plan you can actually follow on a low day rather than only on a good one, and holding it through a flare. Because lupus can involve major organs, we work as part of your care team rather than around it, and your provider coordinates medication changes with your rheumatologist, carefully and never faster than your labs support. Patients often tell us the change they notice first is getting through a day without the fatigue deciding what they are allowed to do.

In our survey of more than 11,000 people following a low-carb high-protein diet, 91% of those with joint and musculoskeletal conditions reported improvement, and 97% of those with autoimmune conditions.² In an independent Harvard Medical School affiliated study, 89% of participants with autoimmune conditions reported their condition resolved or improved.³

Psoriatic Arthritis

What psoriatic arthritis is

Psoriatic arthritis is inflammatory joint disease that develops in some people with psoriasis, and occasionally before any skin involvement appears. Around one in three people with psoriasis will develop it.

It has a distinctive signature. It is often asymmetrical, it frequently hits the joints nearest the fingernails, and it produces two things that are close to diagnostic: a whole finger or toe swelling into a sausage shape, and pain where tendons attach to bone, most often at the heel.

How psoriatic arthritis affects your day

• Joint pain and swelling, often on one side rather than both
• A whole finger or toe swollen rather than a single joint
• Heel pain, or pain at the back of the ankle, that makes walking hard first thing
• Morning stiffness
• Nail changes: pitting, ridging, lifting from the nail bed
• Lower back pain in some people
• Fatigue
• Skin plaques, which may be extensive or barely there

What is driving the pain

• Metabolic inflammation. The link between excess weight and psoriatic arthritis is unusually strong, both for developing it and for how well treatment works. Fat tissue secretes inflammatory signals that feed directly into joint and skin disease, which makes this one of the most responsive conditions on this page to a metabolic intervention.
• Insulin resistance. Commonly present alongside psoriatic disease and part of the same inflammatory picture rather than a coincidence.
• Shared inflammatory pathways. The same signalling drives the skin and the joints, which is why addressing the underlying inflammation tends to improve both together.
• Enthesitis. Inflammation where tendon meets bone is a primary source of pain here and responds differently from joint-lining inflammation, which is part of why psoriatic arthritis can feel undertreated.
• Existing psoriasis. The largest single risk factor, with nail involvement and more extensive skin disease raising the likelihood.

How Revero treats psoriatic arthritis

Psoriatic arthritis is the most metabolically driven condition on this page, and that works strongly in your favour, because metabolism is exactly what our nutrition therapy changes.

This has been tested. In a study of a low-carbohydrate approach in psoriatic arthritis, participants saw significant reductions in pain, in joint and skin disease activity, and in how much the condition interfered with their ability to work, all by week nine, alongside improvements in insulin and lipid levels. A review of low-carbohydrate approaches in inflammatory arthritis found they reduce inflammation and ease joint symptoms across this group of conditions.

Removing the foods provoking immune activity addresses the joints and the skin together, since both run on the same pathways. Lowering carbohydrate reduces insulin resistance and excess weight, which in this condition are active contributors rather than side issues, and raises ketone levels that suppress inflammatory signalling directly.

Your lab panel here is broader than joint inflammation alone. Your provider follows hs-CRP together with fasting insulin and lipids, because watching insulin fall alongside your joint scores is direct evidence the mechanism is working. You track joints, morning stiffness, and skin daily, so both halves of the condition are visible.

Your coach supports the nutrition plan and the weight change that usually accompanies it, which in psoriatic arthritis improves how well your other treatments work too. As your scores improve, your provider can begin reducing medication, gradually and with your consent.

In our survey of more than 11,000 people following a low-carb high-protein diet, 91% of those with joint and musculoskeletal conditions and 96% of those with skin conditions reported improvement.² In an independent Harvard Medical School affiliated study, 96% of participants with musculoskeletal conditions reported their condition resolved or improved.³

Fibromyalgia

What fibromyalgia is

Fibromyalgia is widespread pain lasting months, alongside fatigue, unrefreshing sleep, and difficulty thinking clearly. It is diagnosed clinically, because nothing shows up on imaging or standard blood work.

That last fact has caused an enormous amount of harm. A great many people with fibromyalgia have been told, sometimes explicitly, that because nothing is visible nothing is really wrong. That is not what normal tests mean.

What is happening in fibromyalgia is that the pain processing system itself is amplifying. Nerve pathways transmit pain signals more readily, the brain regions handling them are more reactive, and the thresholds that should filter ordinary sensation are set too low. It is a real, physical, measurable phenomenon. It simply does not show up on the tests most people are given.

There is also a documented inflammatory component. People with fibromyalgia consistently show elevated inflammatory messengers, particularly IL-6, which is one of the same cytokines that amplifies pain signalling.

How fibromyalgia affects your day

• Widespread pain, often described as aching, burning, or deep soreness
• Waking unrefreshed no matter how long you slept
• Fatigue that makes ordinary tasks feel disproportionate
• Difficulty concentrating, word-finding problems, mental fog
• Heightened sensitivity to touch, temperature, light, and noise
• Stiffness, usually worst in the morning
• Digestive symptoms, which overlap with irritable bowel syndrome in most patients
• Headaches
• Low mood and anxiety, which are consequences of living like this as much as anything else

What is driving the pain

• An amplified pain system. The central mechanism. Once pain processing is turned up, the amount of tissue damage needed to produce severe pain drops to almost nothing.
• Low-grade inflammation. Elevated IL-6 and related messengers are consistently found in fibromyalgia, and these are direct pain sensitisers. This is the most addressable driver, and it is the one our approach targets.
• Gut involvement. Digestive symptoms and altered gut bacteria are present in the majority of fibromyalgia patients. That overlap is not incidental, and it is why dietary approaches work here at all.
• Unrefreshing sleep. Deep sleep is disrupted in fibromyalgia, and deep sleep is when pain thresholds reset. This is arguably the most important loop to break.
• Excess weight. Associated with more severe fibromyalgia symptoms, through both mechanical load and inflammatory signalling.
• Blood sugar instability. Swings in glucose produce fatigue, poor sleep, and cognitive fog, all of which compound the underlying condition.

How Revero treats fibromyalgia

Fibromyalgia is often described as untreatable by diet. The research says otherwise, and it says so in a way that is unusually specific to what we do.

In a randomized controlled trial in fibromyalgia, patients followed an anti-inflammatory diet excluding gluten, dairy, added sugar, and ultra-processed foods, combined with a reduction in fermentable carbohydrate in the first month. The control group received general healthy eating advice. The intervention group improved significantly on patient-reported outcomes, and the benefit held regardless of age, how long they had been ill, or how much weight or body fat they lost. In other words, the improvement was not simply a consequence of losing weight.

That intervention is close to what your Revero plan does. Separately, a study of a very low-carbohydrate approach in women with fibromyalgia followed a ketogenic phase with progressive reintroduction of carbohydrate and measured improvements in fibromyalgia impact, mood, and quality of life. That two-phase design, elimination then reintroduction, is exactly the structure your care team uses.

Mechanistically, the chain is clear. Lower the inflammatory messengers and you lower the sensitisation of a pain system that is already turned up too high. Randomized trial evidence shows low-carbohydrate diets significantly lower TNF-alpha and IL-6, and IL-6 is one of the cytokines specifically elevated in fibromyalgia. Stabilising blood sugar addresses the fatigue and fog, and improving sleep begins to reset pain thresholds directly.

Your provider runs a full panel at baseline, which matters here because thyroid disease, vitamin D deficiency, and anemia all mimic or worsen fibromyalgia and are worth identifying before anything else. From there you track pain, fatigue, sleep quality, and mood daily on validated instruments.

Your coach is the difference on this condition more than almost any other. Fibromyalgia is unpredictable, energy is limited, and a plan that assumes you can cook from scratch every evening will fail. Your coach builds around low-energy days, uses better days for preparation, and keeps the reintroduction phase moving so you find your specific triggers rather than staying restricted indefinitely.

Among Revero patients who entered the program with the lowest quality-of-life scores, the pain domain improved by 5.4 points and physical functioning by 8.7 points within three months, alongside an 11.3 point improvement in energy and fatigue.¹ In our survey of more than 11,000 people following a low-carb high-protein diet, 91% of those with joint and musculoskeletal conditions reported improvement.²

Chronic Back Pain

What chronic back pain is

Back pain becomes chronic when it persists beyond about three months, past the point where any original injury would have healed. It is the leading cause of disability worldwide, and it is one of the most poorly explained conditions in medicine.

Part of the reason is that imaging correlates badly with pain. Plenty of people with significant disc degeneration on a scan have no pain at all, and plenty of people in severe daily pain have scans that look ordinary for their age. If you have been shown an image and told it does not explain your symptoms, that is a limitation of the image, not of your account.

What the research increasingly points to instead is a combination of inflammation and an amplified pain system, both of which are influenced by metabolic health.

How chronic back pain affects your day

• Persistent aching, stiffness, or burning in the lower or mid back
• Pain that worsens with sitting, standing, or a particular position you cannot avoid
• Morning stiffness, and difficulty getting going
• Pain radiating into the hip, buttock, or leg
• Difficulty lifting, bending, or carrying
• Interrupted sleep from pain or from struggling to get comfortable
• Reduced activity, and the loss of strength that follows from it
• The mental weight of pain that has no clear end date

What is driving the pain

• Inflammatory messengers. TNF-alpha and IL-6 are found in degenerating discs and are established pain generators. A review of inflammatory biomarkers in back pain found both correlate directly with how intense and how frequent people’s pain is. This is a driver that responds to diet.
• Metabolic inflammation and disc health. A review in Nature Reviews Rheumatology describes disc degeneration as an immunometabolic process, and identifies it as a common comorbidity of obesity and type 2 diabetes, driven by inflammatory signals released from fat tissue that break down connective tissue.
• Insulin resistance. Analysis of more than 41,000 people in a national health survey found insulin resistance and visceral fat were both associated with low back pain, independent of mechanical load.
• Mechanical load. Real, and worth addressing, but rarely the whole story. It is why weight loss usually helps more than the change in load alone would predict.
• An amplified pain system. Central sensitization is well documented in chronic back pain and explains pain that persists after healing and spreads beyond where it began.
• Sleep disruption and deconditioning. Both self-reinforcing. Pain reduces sleep and movement, and less of either raises pain.

How Revero treats chronic back pain

Most back pain treatment addresses either the mechanics or the pain signal. Very little of it addresses the inflammatory and metabolic layer underneath, which is where a substantial part of chronic back pain actually lives.

That layer is where we work. Randomized trial evidence shows low-carbohydrate diets significantly lower TNF-alpha and IL-6, the two inflammatory messengers found in degenerating discs and shown to correlate directly with pain intensity. Reducing insulin resistance and visceral fat addresses the metabolic contribution documented in large population analyses and in the immunometabolic model of disc degeneration. And weight loss reduces mechanical load at the same time, so you are addressing the problem from two directions rather than one.

Removing the foods that provoke an immune response adds a further layer, and the improvement in sleep that our patients typically report early on begins to unwind the loop where poor sleep lowers your pain threshold the following day.

Your provider runs a full baseline panel including fasting insulin, hs-CRP, lipids, and vitamin D, which is worth knowing because deficiency is common and independently associated with chronic musculoskeletal pain. Those repeat at three, six, and twelve months. You track pain severity, difficulty walking, and difficulty lifting daily, so function is measured rather than just how much it hurts.

Your coach works on the plan and on rebuilding activity at a pace that does not set you back, which matters because the fear of triggering a flare is often what keeps people deconditioned. As your pain and function improve, your provider can review any pain medication you are taking, gradually and with your consent.

Among Revero patients who entered the program with the lowest quality-of-life scores, the pain domain improved 5.4 points, physical functioning 8.7 points, and the degree to which physical health limited work and daily activities improved 15.8 points, all within three months.¹ In an independent Harvard Medical School affiliated study, 96% of participants with musculoskeletal conditions reported their condition resolved or improved.³

How your plan actually works

The first phase finds your triggers

We start by removing the most common inflammatory triggers together, and lowering carbohydrate. Not because you will avoid all of it forever, but because while you are eating all of it there is no way to tell which parts are contributing to your pain.

No blood test answers this reliably. Food sensitivity panels are heavily marketed and are not dependable. Structured elimination followed by careful reintroduction is the most accurate method available, and your care team runs it with you rather than handing you a list.

Then you get your foods back

Once your pain, stiffness, and markers have improved, your care team adds foods back one at a time while you keep tracking daily. No flare, and it stays. Symptoms return, and you have learned something specific and permanent about your own body.

Most people finish eating considerably more variety than they expected, knowing exactly which few things set them off. In a condition where you have probably spent years unable to predict a bad day, that predictability is worth a great deal on its own.

The plan moves with you

Progressing well, and your care team widens it. In a flare, they tighten it. Struggling with it, and you tell your coach and it changes. A plan that assumes good energy and working hands every day is a plan you will abandon, so we build one around the days you actually have.

Medications

You do not stop anything to start with us. You begin your nutrition therapy while continuing everything you currently take, and nothing changes unless your provider recommends it and you agree.

As your pain, function, and inflammatory markers improve, reducing medication becomes a real possibility. Revero uses a structured, stepwise process run by your provider, monitored with lab work and daily tracking, and coordinated with your rheumatologist or specialist. Several of these medications need careful tapering, and having a provider watching your labs and symptoms is precisely what makes reducing them safe.

Many of our patients get there. It happens gradually, with data, and with a physician making the call.

Results

Across all conditions, Revero patients saw hs-CRP, a standard marker of systemic inflammation, improve 25% in three months. Among patients with rheumatoid arthritis, disease activity improved 40.5%.¹

The quality-of-life data is where this page’s results live. Among patients who entered the program with the lowest baseline scores, within three months the pain domain improved 5.4 points, physical functioning 8.7 points, energy and fatigue 11.3 points, and the extent to which physical health limited their work and daily activities improved 15.8 points. The people carrying the heaviest burden at the start improved the most.¹

In our survey of more than 11,000 people following a low-carb high-protein diet, 91% of those with joint and musculoskeletal conditions reported improvement, and 79% of those taking medications reported reducing or eliminating them.² In an independent Harvard Medical School affiliated study, 96% of participants with musculoskeletal conditions reported their condition resolved or improved, and 95% reported their overall health improved.³